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It is understood that anabolic steroids display a very poor percentage of survivability through liver metabolism when ingested orallyand thus were thought to be not effective methods of delivery of muscle mass and strength. However, a series of studies conducted in the 1980s has shown that oral DHT administration to mice results in increased muscle mass and strength within 3 days, with significant decreases in liver disease mortality compared to the vehicle-administered group. Furthermore, the acute toxicity and muscle damage seen with oral DHT have not been previously reported, resulting in much larger daily oral doses of DHT than are typically used for medical purposes (25) but in most cases would probably not cause noticeable effects.In animal studies using a variety of protocols and in vivo model systems, oral DHT administration has also demonstrated a marked and significant increase in muscle mass and strength in laboratory animals of high growth factor activity, including steroid-induced growth hormone (GH), and DTH-treated and DHT-free mice (26, 27) and has also shown a direct correlation between anabolic steroid exposure and muscle mass and strength enhancements in rodents in vivo as evidenced by improved motor, skeletal, and neurological parameters (26-29, 30).Despite the well-documented muscle-growth promoting pharmacological effects of anabolic steroids and their use as therapeutic compounds, significant questions still remain regarding their safety in humans. Some studies have found that DHT has been found in plasma to be an effective treatment of Parkinson's disease and other neurodegenerative diseases, which are associated with abnormal accumulation of α-synuclein (31, 32), a known marker of neuronal death in the brain (33). However, other studies did not find DHT in plasma to be associated with either neurodegeneration (34), stroke (35), or Alzheimer's disease (36), although it was found in some studies in several models of Parkinson's disease as well as in the brains of animals with Parkinson's disease but found to be significantly elevated in some animals (37). In addition, DHT was not found to be acutely or persistently toxic in experimental models of Alzheimer's disease or stroke (38-40). Thus, it is possible that a protective effect of anabolic steroids mediated by increased levels of α-synuclein in the brain may not be a direct result of the observed positive therapeutic effects, but may be due to the increased sensitivity of the brain to neurotoxic agents. The role of α-synuclein in memory and learning has recently been investigated (41, 42), and it is possible that, although higher levels of DHT in plasma may be expected to have some roleSimilar articles: